[Express] “Fukushima university student got thyroid cancer”

Introducing important tweets as [Express] for simultaneous updates.

Fukushima university student got thyroid cancer. Some of them are having unidentified cancer too. Doctor says the causality is not known.

I heard someone in Fukushima university got thyroid cancer too, but it can’t be proven to have something to do with radiation exposure. I heard the story of a baby who had the heart stopped in pregnancy. It doesn’t have media coverage but Fukushima people know that.



Français :

[Express] “un étudiant de l’université de Fukushima a un cancer de la thyroïde”

Présentation des tweets importants sous [Express] pour mises à jour simultanées.

un étudiant de l’université de Fukushima a un cancer de la thyroïde. Certains d’entre eux ont aussi des cancers non identifiés. Le médecin dit que la causalité n’est pas connue.

J’ai entendu dire que quelqu’un de l’université de Fukushima a un cancer de la thyroïde aussi mais on ne peut pas prouver que ce soit lié à l’exposition à la radioactivité. J’ai entendu l’histoire d’un bébéb qui a fait un arrêt cardiaque pendant la grossesse. Ce n’est pas couvert par les médias mais les gens de Fukushima le savent.





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5 Responses to “[Express] “Fukushima university student got thyroid cancer””

  1. jec says:

    People with Thyroid cancer can have the tumor”fingerprint” tested for radiation. They can tell if caused by radiation exposure or fallout. Will check and see where and how to do this test or if its available yet. Anyone with cancers should ask to have samples saved frozen for later research (THEIRS!) if they can. It could be used to make TEPCO pay for damage to health..or the government. If there is not health insurance to pay for these cancers..then the reponsible party needs to provide the coverage.

  2. jec says:


    Fingerprint of radiation exposure discovered in thyroid cancer
    Neuherberg,- Scientists from the Helmholtz Zentrum München have discovered a genetic change in thyroid cancer that points to a previous exposure of the thyroid to ionising radiation. The gene marker, a so-called „radiation fingerprint“ was identified in papilliary thyroid cancer cases from Chernobyl victims, but was absent from the thyroid cancers in patients with no history of radiation exposure. The results are published in the current issue of PNAS.

    Fingerprint of radiation exposure discovered in thyroid cancerThe research team, led by Prof. Horst Zitzelsberger and Dr. Kristian Unger from the Radiation Cytogenetics Unit of the Helmholtz Zentrums München, in collaboration with Prof. Geraldine Thomas, Imperial College London, studied thyroid cancers from children exposed to the radioiodine fallout from the Chernobyl nuclear reactor explosion. The team compared the genetic information from these tumours to that found in the same type of tumour that arose in children born more than one year after the explosion, after the radioactive iodine had decayed away. The number of copies of a small fragment of chromosome 7 was found to be increased only in the tumours from the irradiated children, establishing this as one of the first genetic markers that indicate a radiation aetiology of cancer.

    This breakthrough is the first time since the reactor accident in 1986 that scientists have been able to discriminate between the cancers caused by the radioactive contamination and those that arise naturally. Prof. Zitzelsberger ascribes the success of this study to the careful collection, documentation and storage of thyroid cancers from the Chernobyl region in the Chernobyl Tissue Bank. He noted that this unique collection of materials made it possible for the team to compare for the first time tumours from children of the same age and regional background. The availability of the genetic marker, according to Prof. Zitzelsberger, will improve both the clinical diagnosis of thyroid cancer and our understanding of how radioactive iodine causes the disease to develop. In future studies funded by EURATOM in the project „EpiRadBio“ the group will extend the study to determine if the genetic fingerprint is able to indicate the level of radiation exposure that is required to cause the cancer.

  3. jec says:

    Wonder if the below research professor would like samples from Japanese childen to test?!! It something anyway. If the DNA change in the thyroid cancer can be traced to ionizing radiation-thats a huge step.

    Prof. Dr. Horst Zitzelsberger, Abteilung für Strahlenzytogenetik, Helmholtz Zentrum München – Deutsches Forschungszentrum für Gesundheit und Umwelt (GmbH), Ingolstädter Landstraße 1 85764 Neuherberg. Phone.: + 49 89-3187-3421 .
    Email: zitzelsberger@helmholtz-muenchen.de

  4. jec says:

    Am posting all this technical content because maybe someone in Japan can use to help citizens. Note it is a DNA test used to find the chromosombe band gain, 7q11. Since I am not a scientist with this topic, hope someone is who can pursue.


    Gain of chromosome band 7q11 in papillary thyroid carcinomas of young patients is associated with exposure to low-dose irradiation

    Julia Heßa,
    Gerry Thomasb,
    Herbert Braselmanna,
    Verena Bauera,
    Tatjana Bogdanovac,
    Johannes Wienbergd,
    Horst Zitzelsbergera, and
    Kristian Ungerb,1

    Author Affiliations

    Edited* by Janet D. Rowley, University of Chicago, Chicago, IL, and approved April 26, 2011 (received for review November 21, 2010)


    The main consequence of the Chernobyl accident has been an increase in papillary thyroid carcinomas (PTCs) in those exposed to radioactive fallout as young children. Our aim was to identify genomic alterations that are associated with exposure to radiation. We used array comparative genomic hybridization to analyze a main (n = 52) and a validation cohort (n = 28) of PTC from patients aged <25 y at operation and matched for age at diagnosis and residency. Both cohorts consisted of patients exposed and not exposed to radioiodine fallout. The study showed association of a gain on chromosome 7 (7q11.22–11.23) with exposure (false discovery rate = 0.035). Thirty-nine percent of the exposed group showed the alteration; however, it was not found in a single case from the unexposed group. This was confirmed in the validation set. Because only a subgroup of cases in the exposed groups showed gain of 7q11.22–11.23, it is likely that different molecular subgroups and routes of radiation-induced carcinogenesis exist. The candidate gene CLIP2 was specifically overexpressed in the exposed cases. In addition, the expression of the genes PMS2L11, PMS2L3, and STAG3L3 correlated with gain of 7q11.22–11.23. An enrichment of Gene Ontology terms “DNA repair” (PMS2L3, PMS2L5), “response to DNA damage stimulus” (BAZ1B, PMS2L3, PMS2L5, RFC2), and “cell–cell adhesion” (CLDN3, CLDN4) was found. This study, using matched exposed and unexposed cohorts, provides insights into the radiation-related carcinogenesis of young-onset PTC and, with the exposure-specific gain of 7q11 and overexpression of the CLIP2 gene, radiation-specific molecular markers.



    1To whom correspondence should be addressed. E-mail: unger@helmholtz-muenchen.de.

    Author contributions: G.T., H.Z., and K.U. designed research; J.H., V.B., T.B., J.W., and K.U. performed research; H.B. contributed new reagents/analytic tools; J.H., G.T., H.B., and K.U. analyzed data; and J.H., G.T., V.B., H.Z., and K.U. wrote the paper.

    The authors declare no conflict of interest.

    *This Direct Submission article had a prearranged editor.

    This article contains supporting information online at http://www.pnas.org/lookup/suppl/doi:10.1073/pnas.1017137108/-/DCSupplemental.

  5. Preco says:

    Question for 1 mil $

    WHat is cause for thyiriod cancer ?

    1. Cesium
    2. Radiation
    3. Fallout
    4. Alpha And Beta Particles

    Correct answer is ….. ALL FOUR

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